2 edition of Urea retention in experimental oxalate nephritis found in the catalog.
Urea retention in experimental oxalate nephritis
J. Shaw Dunn
|Statement||by J. Shaw Dunn, Albert Haworth and Nora A. Jones.|
|Contributions||Haworth, Albert., Jones, Nora A.|
|The Physical Object|
|Pagination||p. 377-400 :|
|Number of Pages||400|
Start studying Nephritis. Learn vocabulary, terms, and more with flashcards, games, and other study tools. Search. Create. proteinuria, salt and water retention, and evidenced by azotemia (abnormally high levels of nitrogen wast products in blood), occurring days after infection. Urea nitrogen, the end product of protein. Acute kidney injury (AKI) is characterized by a rapid (hours to weeks) decline in the glomerular filtration rate (GFR) and retention of nitrogenous waste products such as blood urea nitrogen (BUN) and creatinine. In recent years, it has been recognized that the time-honored term acute renal failure (ARF).
Full text of "The clinical examination of urine: with an atlas of urinary deposits" See other formats This is a digital copy of a book that was preserved for generations on library shelves before it was carefully scanned by Google as part of a project to make the world's books discoverable online. a form of nephritis that involves primarily the glomeruli; also known as Bright's disease white/yellow crystals of urate on the skin Urea compounds and other waste products of metabolism that cannot be excreted by the kidneys into the urine are excreted through the small superficial capillaries into the skin, where they collect on the.
Kidney International (KI) is the official journal of the International Society of Nephrology. Under the editorial leadership of Dr. Pierre Ronco (Paris, France), KI is one of the most cited journals in nephrology and widely regarded as the world's premier journal on the development and consequences of kidney disease. Uric acid is the end product of purine metabolism in humans. In animals other than mammals uric acid undergoes further degradation because of the activity of enzymes such as uricase, allantoinase and allantoinase; in some species the urea that is formed is further hydrolyzed to ammonia and CO2 by the urease of intestinal bacteria (1).Cited by: 1.
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Urea retention, in the sense of a relatively increased concentration in the blood, is the result of increased resistance to the excretion of urea through the kidneys. The relatively increased concentration of urea in the blood overcomes the increased resistance to excretion, and the organism is thereby maintained in nitrogenous equilibrium.
by: 4. In experimental nephritis caused by cantharidin, arsenic, diphtheria toxin, and potassium chromate, in addition to the retention of the non-protein and urea nitrogen and of the chlorides in blood, an acidosis occurs. Sodium bicarbonate given by the stomach has the power of diminishing the acidosis in these types of by: 7.
An acute oxalate nephritis leads to marked retention of urea, but even when the urea concentration in the blood is at its highest point no albumen may be present in the urine urine Subject Category: Miscellaneous see more details.
It is important to bear this in mind before excluding nephritis in accidental haemorrhage. " by: 1. Notice: Except where noted, spectra from this collection were measured on dispersive instruments, often in carefully selected solvents, and hence may differ in detail from measurements on FTIR instruments or in other chemical environments.
More information on the manner in which spectra in this collection were collected can be found here. Notice:. In experimental nephritis caused by cantharidin, arsenic, diphtheria toxin, and potassium chromate, in addition to the retention of the non-protein and urea nitrogen and of the chlorides in blood, an acidosis occurs.
Sodium bicarbonate given by the stomach has the power of diminishing the acidosis in these types of nephritis. by: 7. UREA: ITS DISTRIBUTION IN AND ELIMINATION FROM THE BODY.
BY E. MARSHALL, JR. AND DAVID M. DAVIS. (JFrom the Laboratory of Physiological Chemistry and the Hunterian Laboratory of Experimental Pathology, Johns Hopkins University.) (Received for.
SUMMARY A comparative study of the ratio of the rate of excretion to concentration in the blood for the nitrogenous waste products, shows that in experimental uranium nephritis creatinine is eliminated with slightly greater difficulty than urea in mild cases of nephritis; whereas in the severer types, the urea retention is more marked than that of by: 2.
General Conclusions. A fatal nephritis may result from the action of a, filter-passing virus in the absence of any other variety of parasite. Such a virus may cause death with urea retention, although the virus was obtained from diseased nervous tissue, and although the portal of entry was by the central nervous system.
by: The presence of large quantities of oxalate in the kidneys and urine of cats deficient in vitamin B 6, in amounts sufficient to cause marked renal damage, is attributable to excessive endogenous formation of by: In experimental nephritis induced by tartrate a marked dilution of the blood may occur (5).
It is likewise well known that in experimental nephritis due to administration of uranium salts there is a marked retention of non-protein nitrogen and urea (6).Cited by: 2. In such cases the blood urea mounted rapidly; the urea clearance fell; and death occurred within about 2 weeks.
A milder nephritis of the chronic type was induced by giving smaller quantities of anti-kidney serum in either single or divided by: For the Love of Physics - Walter Lewin - - Duration: Lectures by Walter Lewin.
They will make you ♥ Physics. Recommended for you. Oxalate nephropathy with a granulomatous lesion due to excessive intake of peanuts Article in Clinical and Experimental Nephrology 12(4). Kidney stone disease is a crystal concretion formed usually within the kidneys. It is an increasing urological disorder of human health, affecting about 12% of the world population.
Lemon juice has protective activity in a rat urolithiasis oxalate nephritis caused by et hylene grycol. The concentration of urea was significantly (p.
In rats treated with lemon juice, we attribute the lower serum creatinine and urea levels to an enhanced GFR and the anti-lipid peroxidative property of lemon juice. As commended, the lithogenic effects of EG must be mainly attributed to the oxidative damage caused by the high level of oxalate generated by this by: Regulate Oxalate Metabolism: Hyperoxaluria is a most significant risk factor in the pathogenesis of renal stone.
It has been reported that oxalate play an important role in stone formation and has about fold greater effect than urinary calcium. Increased oxalate concentration is responsible for precipitation and deposition of CaOx crystals.
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Journal of Evolutionary Biochemistry and Physiology Journal of Experimental. The protective effects of anticoagulants in nephrotoxic nephritis in rabbits have been studied, using various doses of heparin and defibrination with ancrod. Massive doses of heparin ( units/kg/day) were required before significant reduction in glomerular fibrin deposition, extracepillary cell proliferation and urea retention by: Nephro- protective effects of Arabian medicinal plants (part 1).
experimental oxalate. rapid decline in renal function resulting in abnormal retention of serum creatinine and blood urea. texts All Books All Texts latest This Just In Smithsonian Libraries FEDLINK (US) Genealogy Lincoln Collection. Books to Borrow. Top Full text of "Renal diseases: a clinical guide to their diagnosis and treatment".illustrations of different types of retention.
The few cases other t,han nephritis are tabulated for comparison. Case 1, suffering from mercuric chloride poisoning, showed a very pronounced retention of all the non-protein nitrogenous constituents, the figures for non-protein nitrogen, urea. ); since alcohol extraction would yield poor results, I chose to use Oxalic acid(as I have two pounds of this stuff) to precipitate out the urea in the form of urea oxalate.
The next step of this procedure would be to add it to a sodium bicarbonate solution, leaving the urea in solution and precipitating some some sodium oxalate.